Cut mix 150

Testosterone Propionate is one of the many esterified variants of Testosterone available. It is an injectable compound with a slower rate of release than un-esterified Testosterone, but a faster rate of release than all other esterified forms commonly available. This is due to the larger Propionate ester attached to the Testosterone molecule. This augments the release rate and half-life of Testosterone to that of a faster release than other common esterified variants, such as Testosterone Enanthate or Testosterone Propionate. The majority of Testosterone products that have been designed are single products that contain a single esterified form (such as this one), as opposed to Testosterone products which consist of a blend of several different esterified variants in the liquid (such as Sustanon 250, for example). Testosterone that is un-esterified holds a very short half-life, making its use very inconvenient and impractical

The specific name for Masteron is actually Dromostanolone (Drostanolone was a slightly more abbreviated name given to the compound shortly after its release). As mentioned previously, it is a modified derivative of DHT (Dihydrotestosterone), placing it into the family of DHT-derivatives and analogues. Masteron is a modified form of DHT, where a methyl group at the 2nd carbon (known as carbon alpha) atom. This modification is what is known to be responsible for the slight anabolic strength increase in comparison to Testosterone. This methyl group addition increases the anabolic strength by way of granting Masteron an increased resistance to being metabolized into inactive metabolites by the enzyme 3-hydroxysteroid dehydrogenase. This enzyme is present in large quantities in muscle tissue, and is the enzyme that serves to metabolize any DHT that enters muscle tissue into two inactive metabolites: 3-Alpha Androstanediol and 3-Beta Androstanediol that are non-anabolic what so ever in muscle tissue. This is therefore the reason as to why DHT is not anabolic in muscle tissue at all, and many chemists and biologists believe that if the enzyme 3-hydroxysteroid dehydrogenase did not exist in muscle tissue, that DHT would actually be a very potent and powerful anabolic steroid.

Trenbolone is essentially a derivative of Nandrolone with some very significant differences in its chemical properties and strength. Trenbolone and its parent hormone Nandrolone both belong to a class/category of anabolic steroids known as 19-nor compounds, or 19-nors (short for 19-nortestosterone). 19-nor anabolic steroids are labeled as such because they lack the 19th carbon on their structure – this carbon exists on Testosterone and all other anabolic steroids with the exception of 19-nor compounds, such as Nandrolone and Trenbolone. This significantly changes around the properties of an anabolic steroid and makes it a Progestin, which will be discussed further shortly. In Trenbolone, this missing carbon atom at the 19th position (which is in reality a whole methyl group) is replaced by double-bonds between the two carbon atoms that the 19th carbon was originally bound with (this differs from Nandrolone where the lacking 19th carbon is simply replaced with a hydrogen atom instead of double-bonds in Trenbolone’s case). This lack of a 19th carbon is what makes 19-nor compounds very resistant to the aromatase enzyme and therefore very resistant to any estrogen conversion – however, this is not the whole story for Trenbolone when it comes to aromatization. Trenbolone also contains modifications at carbons 19 and 11, where one hydrogen atom was removed from each carbon so that carbons 19 and 11 become double-bonded with their neighboring carbon atoms in their respective cycloalkane rings. It is these additional modifications of double-bonds at carbon 19 and 11 that grant Trenbolone to be not just resistant to aromatization, but to become completely immune to it and be unable to interact what so ever with the aromatase enzyme. These modifications are also responsible for Trenbolone’s extremely enhanced andrognic strength (its ability to bind at a much greater strength to the androgen receptor) and its ability to remain highly resistant to metabolic breakdown in the body.